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These changes were not observed in the placebo group

Cancer 127 (8), 14031410

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pylori could colonize gastric epithelial cells due to its main components enabling it to survive in an acidic stomach environment and attach to host cells through the interaction of microbial adhesins with host cell receptors through flagellate-mediated motility

In summary, the development of pharmacological agents that promote itaconate production could potentially mitigate pathological inflammatory responses and prevent severe lung injury associated with TB progression

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