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Insulin plays a neuroprotective role in AD by enhancing A clearance through the upregulation of neprilysin (NEP) and IDE while also stimulating gluconeogenesis following ischemic stroke 67

Across all disease phases of various ADs, Clostridium -based interventions can serve as adjuvants: In the acute phase, they assist conventional therapies in alleviating inflammation by reinforcing the intestinal barrier and suppressing pro-inflammatory pathways

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GSH deficiency inhibits ATP7A and ATP7B resulting in the intracellular accumulation of copper [136]

These receptors reduce the response of GABA receptors, and the net result is the removal of limiters on dopamine release