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However, considering the role of oxidative stress in DPN pathology, as well as identified mechanisms by which ALA may alleviate DPN symptoms, it would seem reasonable to assume that regular intake of ALA (e.g., 300 600 mg/d) may offer some protection against developing DPN symptoms, even when used as a monotherapy

These previous studies indicated that Shh/Gli-1 signaling may represent a potential therapeutic target for the regulation of microglial activation in central nervous system diseases

A., Vivas, E