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This is more physiologically appropriate and likely explains why the combination of PTD-DBM (upstream release) with valproic acid (downstream activation) produces synergistic rather than merely additive effects

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The VPA-induced ASD model group showed significantly higher dissimilarity from the control group, indicating profound dysbiosis

This lack of effects due to higher doses of CGV could results from a saturation activity at lower doses, although, we did not assess the neuroendocrine effects of CGV